This is a placeholder text
Group text
by Ibrahim on 01 October 2013 - 16:10
Thank you Nedev, I appreciate that
by Nedev385 on 02 October 2013 - 01:10
I also had a problem with one of my dogs.It is my pleasure to help you.More serious problem after the disease does not have a heart attack and die after 5-6 months.I have seen such cases.Do not load 6-7 months and the food is good for his heart (without much fat).Regards.
by 123lyn45 on 26 November 2013 - 19:11
I have noticed people on this forum are saying that if a pup has parvo he then becomes immune this is totally incorrect, and the virus can re-occur.CANINE PARVOVIRUS
Nature of the disease
Canine parvovirus is a highly contagious infection of dogs caused by a Parvovirus and characterised by sudden onset of vomiting, diarrhoea, dysentery, depression, anorexia, pyrexia, severe dehydration, leucopaenia and death. Sudden death from myocarditis occurs in very young puppies.
Classification
SPC List D disease
Susceptible species
Dogs. Canine parvovirus is primarily a disease of young dogs. Rottweilers, American Pit Bull Terriers, Doberman, Pinschers and German Shepherd are more susceptible.
Distribution
Canine parvovirus appeared suddenly right around the world, including Australia and New Zealand, in 1978.
Clinical signs
Clinical signs are very variable with puppies the most severely affected.Mortality rates can be high.
There are two main clinical syndromes associated with canine parvovirus infection — enteric form, and myocardial or heart form
Enteric form
• Sudden depression
• Loss of appetite
• Fever especially in puppies
• Vomiting and diarrhoea (in up to 50% of cases, diarrhoea may be haemorrhagic)
• Rapid dehydration
• Shock and death (in acute cases)
If the dog survives the enteritis and dehydration, it becomes immune, but recovery may be slow.
Myocardial form or heart form
The first form is associated with sudden death. The puppy appear normal, then gasps, mucous membranes turn pale and then cyanotic, with death occurring in about two hours due to acute non-suppurative myocarditis.
The heart form is associated with congestive heart failure. There is slower onset of clinical signs:
• Dyspnoea
• Depression
• Cough
• Ascites
• Death typically at 12–16 weeks of age
Post-mortem findings
In the enteric form the following are seen:
• Lower and middle small intestine is dilated — contents are watery and flocculent
• Bone marrow is depleted
In the myocardial form the following is seen:
• Lungs are heavy and oedematous, grey-pink in colour with focal congestion
• Heart is dilated will ill-defined pale areas in the myocardium
• May be haemorrhages in the pancreas
• Clear watery fluid in the thorax and abdomen
• Pale, flabby heart with myocardial fibrosis in more chronic cases
Differential diagnosis
Other cause of enteritis to be considered include:
• Canine distemper
• Infectious canine hepatitis
• Canine coronavirus
• Salmonellosis
• Campylobacteriosis
• Haemorrhagic gastro-enteritis
• Poisonings
Other cause of myocarditis include:
• Canine distemper
• Infectious canine hepatitis
• Canine herpesvirus
• Streptococcal infection
• Congenital heart anomalies
Specimens required for diagnosis
Canine parvovirus can be suspected on clinical grounds in severe cases. In less severe cases, laboratory assistance may be required.
Faecal material should be collected from suspect cases for attempted virus isolation and/or virus detection.
At post-mortem, fresh samples of mesenteric lymph nodes, tonsils, small intestine and intestinal contents should be collected and forwarded chilled, on water ice or frozen gel packs, to the laboratory.
Serum samples from recovered or older in-contact animals can be collected for serology.
Transmission
Animals are infected by contact with infected faeces, usually by ingestion — via feeding utensils, licking infected dogs, etc. Virus can survive for up to 12 months in faeces. The virus is also present in urine.
Infection is spread directly and indirectly by anything contaminated by faecal material (shoes, coats, hands, equipment).
Risk of introduction
Canine parvovirus has shown the ability to spread widely and rapidly. The virus can survive for long periods. The disease could be introduced with an infected dog or via clothing or equipment contaminated with faecal material.
Control / vaccines
With the enteric syndrome, supportive therapy is important. Treatment should include electrolyte fluid replacement, gastric sedatives and anti-emetics.
With the myocardial form, treatment is ineffective.
Vaccines are available. If the bitch has been vaccinated, puppies will have some passive immunity that will decline over the first 3 months of age. To be protected, puppies need to be first vaccinated at 6 weeks with repeat doses at 12-14 weeks and 16-18 weeks. Annual boosters are recommended.
For dog breeders, there should be careful attention to hygiene and disinfection of kennels.
References
• Canine Parvovirus, In Merck Veterinary Manual, National Publishing Inc. Eight ed, 1998, Philadelphia, p 285-286
• Parvovirose du chien, In Dictionnaire Pratique de Thérapeutique Canine et Féline, Masson, Forth ed, 1997, Paris, p 401-402
Just wanted to share as a friend has had a breakout and has lost 3 babies, terrible diesease....
Nature of the disease
Canine parvovirus is a highly contagious infection of dogs caused by a Parvovirus and characterised by sudden onset of vomiting, diarrhoea, dysentery, depression, anorexia, pyrexia, severe dehydration, leucopaenia and death. Sudden death from myocarditis occurs in very young puppies.
Classification
SPC List D disease
Susceptible species
Dogs. Canine parvovirus is primarily a disease of young dogs. Rottweilers, American Pit Bull Terriers, Doberman, Pinschers and German Shepherd are more susceptible.
Distribution
Canine parvovirus appeared suddenly right around the world, including Australia and New Zealand, in 1978.
Clinical signs
Clinical signs are very variable with puppies the most severely affected.Mortality rates can be high.
There are two main clinical syndromes associated with canine parvovirus infection — enteric form, and myocardial or heart form
Enteric form
• Sudden depression
• Loss of appetite
• Fever especially in puppies
• Vomiting and diarrhoea (in up to 50% of cases, diarrhoea may be haemorrhagic)
• Rapid dehydration
• Shock and death (in acute cases)
If the dog survives the enteritis and dehydration, it becomes immune, but recovery may be slow.
Myocardial form or heart form
The first form is associated with sudden death. The puppy appear normal, then gasps, mucous membranes turn pale and then cyanotic, with death occurring in about two hours due to acute non-suppurative myocarditis.
The heart form is associated with congestive heart failure. There is slower onset of clinical signs:
• Dyspnoea
• Depression
• Cough
• Ascites
• Death typically at 12–16 weeks of age
Post-mortem findings
In the enteric form the following are seen:
• Lower and middle small intestine is dilated — contents are watery and flocculent
• Bone marrow is depleted
In the myocardial form the following is seen:
• Lungs are heavy and oedematous, grey-pink in colour with focal congestion
• Heart is dilated will ill-defined pale areas in the myocardium
• May be haemorrhages in the pancreas
• Clear watery fluid in the thorax and abdomen
• Pale, flabby heart with myocardial fibrosis in more chronic cases
Differential diagnosis
Other cause of enteritis to be considered include:
• Canine distemper
• Infectious canine hepatitis
• Canine coronavirus
• Salmonellosis
• Campylobacteriosis
• Haemorrhagic gastro-enteritis
• Poisonings
Other cause of myocarditis include:
• Canine distemper
• Infectious canine hepatitis
• Canine herpesvirus
• Streptococcal infection
• Congenital heart anomalies
Specimens required for diagnosis
Canine parvovirus can be suspected on clinical grounds in severe cases. In less severe cases, laboratory assistance may be required.
Faecal material should be collected from suspect cases for attempted virus isolation and/or virus detection.
At post-mortem, fresh samples of mesenteric lymph nodes, tonsils, small intestine and intestinal contents should be collected and forwarded chilled, on water ice or frozen gel packs, to the laboratory.
Serum samples from recovered or older in-contact animals can be collected for serology.
Transmission
Animals are infected by contact with infected faeces, usually by ingestion — via feeding utensils, licking infected dogs, etc. Virus can survive for up to 12 months in faeces. The virus is also present in urine.
Infection is spread directly and indirectly by anything contaminated by faecal material (shoes, coats, hands, equipment).
Risk of introduction
Canine parvovirus has shown the ability to spread widely and rapidly. The virus can survive for long periods. The disease could be introduced with an infected dog or via clothing or equipment contaminated with faecal material.
Control / vaccines
With the enteric syndrome, supportive therapy is important. Treatment should include electrolyte fluid replacement, gastric sedatives and anti-emetics.
With the myocardial form, treatment is ineffective.
Vaccines are available. If the bitch has been vaccinated, puppies will have some passive immunity that will decline over the first 3 months of age. To be protected, puppies need to be first vaccinated at 6 weeks with repeat doses at 12-14 weeks and 16-18 weeks. Annual boosters are recommended.
For dog breeders, there should be careful attention to hygiene and disinfection of kennels.
References
• Canine Parvovirus, In Merck Veterinary Manual, National Publishing Inc. Eight ed, 1998, Philadelphia, p 285-286
• Parvovirose du chien, In Dictionnaire Pratique de Thérapeutique Canine et Féline, Masson, Forth ed, 1997, Paris, p 401-402
Just wanted to share as a friend has had a breakout and has lost 3 babies, terrible diesease....
by greyhoundgirl on 26 November 2013 - 20:11
People keep saying your dog can't get parvovirus once they've had it because it's true. No where in your blurb that you posted mentioned reinfection either.
by SitasMom on 26 November 2013 - 20:11
i'm going with vaccinate as usual, for all the stuff... and then rabies too.
cannot be too careful.
cannot be too careful.
by hexe on 26 November 2013 - 21:11
Simple way to determine if the pup was able to build up a sufficient immune response: have blood drawn and sent out for a parvo titer. If it's below what's considered a protective level, vaccinate against parvo. If it's at a protective level, then it's your call, Ibrahim.
The reason puppies [and children, for that matter] receive a series of vaccinations is because [a] we need to overrride the maternal antibodies--which may still have been in play with the pup in question, hence the somewhat easier recovery than most pups experience from parvo, and [b] the immune system in immature mammals is also immature, and may NOT mount a sufficient defense against a field challenge even if the pup has been vaccinated or had been infected with a disease and then is exposed to the wild virus yet again at a later point in the pup's maturation stages.
Personally, I would never forgive myself if I presumed that the immune response developed by an 8 to 10 week old puppy as the result of field-strain infection turned out to be insufficient, and the pup was lost later on exposure to the virus. But that's what works for me. I also vaccinate my dogs twice a year for leptospirosis, and booster every other year for distemper, parainfluenza, parvo and adenoviruses, along with the triennial rabies booster. And have never had any issues arise as a result of this 'overvaccination', as it's been called. I've also never lost one of my dogs to a viral disease that I could have prevented if I'd ensured that they were able to mount a sufficient immune response against that illness, either. Everyone's got to make their own call for their own animals.
The reason puppies [and children, for that matter] receive a series of vaccinations is because [a] we need to overrride the maternal antibodies--which may still have been in play with the pup in question, hence the somewhat easier recovery than most pups experience from parvo, and [b] the immune system in immature mammals is also immature, and may NOT mount a sufficient defense against a field challenge even if the pup has been vaccinated or had been infected with a disease and then is exposed to the wild virus yet again at a later point in the pup's maturation stages.
Personally, I would never forgive myself if I presumed that the immune response developed by an 8 to 10 week old puppy as the result of field-strain infection turned out to be insufficient, and the pup was lost later on exposure to the virus. But that's what works for me. I also vaccinate my dogs twice a year for leptospirosis, and booster every other year for distemper, parainfluenza, parvo and adenoviruses, along with the triennial rabies booster. And have never had any issues arise as a result of this 'overvaccination', as it's been called. I've also never lost one of my dogs to a viral disease that I could have prevented if I'd ensured that they were able to mount a sufficient immune response against that illness, either. Everyone's got to make their own call for their own animals.
by Q Man on 27 November 2013 - 12:11
Most important thing is to protect puppies when they are young...It will help protect them for their entire lives...I begin giving vaccines at 6 weeks of age...I also give a set of 3 shots (5-1)...then a Rabies Shot at around 16 weeks of age...
I've had good luck with this method and continue to use it until I have better information...I'm also not a proponent of giving ALL vaccines yearly...As you've probably seen...It's now the practice of giving Rabies Shots every 3 years and even longer...So it's getting to be known that not all vaccines are needed yearly...That they protect long after first giving...
~Bob~
I've had good luck with this method and continue to use it until I have better information...I'm also not a proponent of giving ALL vaccines yearly...As you've probably seen...It's now the practice of giving Rabies Shots every 3 years and even longer...So it's getting to be known that not all vaccines are needed yearly...That they protect long after first giving...
~Bob~
by Ibrahim on 27 November 2013 - 12:11
Thanks Bob
by Spooks on 28 November 2013 - 02:11
Q Man
Giving Parvo vaccinations to puppies that young, you run the risk of it being over-ridden by the maternal anti-bodies, thus rendering it ineffective.
Please read a fuller description in my post above.
Giving Parvo vaccinations to puppies that young, you run the risk of it being over-ridden by the maternal anti-bodies, thus rendering it ineffective.
Please read a fuller description in my post above.
by Hundmutter on 28 November 2013 - 03:11
Re: immunity vs re-occurence, didn't I read someplace that Parvo
is like 'flu, the virus mutates ? Maybe that's why some dogs get
it more than once ?
is like 'flu, the virus mutates ? Maybe that's why some dogs get
it more than once ?
Contact information Disclaimer Privacy Statement Copyright Information Terms of Service Cookie policy ↑ Back to top
